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It is important to understand the role of lipoprotein(a) relative to cardiovascular health. Lp(a) has been linked to two functional bodily systems:
Because of that Lp(a) has a direct impact on the development of atherosclerosis. Somehow lipoprotein(a) thickens the blood and stiffens artery walls. More precisely, it is able to bind to fibrin clots in the injured arterial endothelium (the innermost lining of arteries) and deliver cholesterol to that region in order to heal the wound. In this way, Lp(a) contributes to the formation of fatty buildups called atheromas and, as a matter of fact, it becomes a component of the arterial plaque. From that perspective, the role of this little-known killer component of cholesterol in atherogenesis can be both long-term - due to its lipoprotein properties, and short-term - due to its fibrin-binding properties. Further, it may cause the sudden and more dangerous development of arterial blood clots.
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It may be responsible for many of the toxic effects formerly attributed to cholesterol and its LDL subfraction. It may cause the oxidation of LDL and damage the cardiovascular system even more actively in the presence of homocysteine - another risk factor for cardiovascular disease. Without any doubt, elevated Lp(a) levels contribute to:
Lipoprotein(a) has been also suggested to increase the risk of Alzheimer’s disease and dementia, especially in poststroke dementia – a chronic or persistent deterioration of intellectual function and cognitive skills. Symptoms of dementia include memory loss, personality changes, lack of interest in personal care, impaired reasoning ability, and disorientation. Dietary trans fat and soy proteins may increase the body’s production of Lp(a). People with hypothyroidism (underactive thyroid) have often elevated lipoprotein(a) levels.
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Not the wrongly blamed LDL-“bad” cholesterol then – but the Lp(a) and Apo(a) carried by the Lp(a) throughout the body should be considered responsible for the majority of the detrimental effects on the vascular system. According to new evidence the size of apolipoprotein(a) may be of importance in conveying the Lp(a)-related risk. Specifically, the variation in apo(a) isoform size may lead to increased risk of vascular dementia, Alzheimer’s disease, stroke, or all three of them. (Dementia and Geriatric Cognitive Disorders, 18(2):189-196, 2004).
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As standard reference ranges differ dangerously, the healthy levels of Lp(a) should be less than 14 mg/dL. So, if you have not checked your lipoprotein(a) yet do it at your next physical checkup. And do not be afraid to ask your doctor for it! Unfortunately, very few physicians ever check for Lp(a). In heart patients, it should be routinely screened. If your Lp(a) level is over 14 mg/dL you need to take action at once. And if you have high lipoprotein(a) and relatively normal LDL-“bad” cholesterol you can probably benefit from lowering LDL as much as possible first.
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But like other drugs, Niaspan has side effects, not to mention multiple contraindications.
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The general guidelines provided by the N.C.E.P (National Cholesterol Education Program) do not address the risk factors unique to Asian Indians such as:
As a powerful independent risk factor for premature fatty deposition and thrombosis (blood clotting) in the blood vessels, lipoprotein(a) is also responsible for the high failure rate of balloon angioplasty and bypass surgery. 30 percent Asian Indians have Lp(a) levels above 30mg/dL – being at significantly increased risk of coronary artery disease – compared to Caucasians (19 percent) and Mexican Americans (only 8 percent). Nearly 95 percent of lipoprotein(a) is genetically determined (so it runs in families) and adult levels are reached by the first year of life. If either of the parents has a high level of Lp(a) there is a strong probability of their children developing premature coronary artery disease because their Lp(a) levels are identical to that of their parents. Therefore, these children should be screened at the earliest age possible, so prevention could be offered to halt the progress and change the natural course of heart disease. This explains why the disease process starts 15-20 years earlier than when it is caused by conventional risk factors like smoking and hypertension (high blood pressure). It has been said that very little could be done about Lp(a) because its blood concentrations are determined by genetics. However, this statement may be true only from pharmacological point of view. The lipoprotein(a) modification is difficult – but it can be done with targeted nutritional supplementation providing such nutrients as L-Cysteine, nicotinic acid (vitamin B3), tocotrienols (vitamin E), ascorbic acid (vitamin C), and other vital lipid-modulating nutrients and phytonutrients.
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There have been medical studies on the beneficial effect of nutritional supplementation on triglyceride levels; it appears people with high triglycerides do benefit from dietary supplements. Nutritional factors - naturally occurring substances, not drugs whose substances are foreign to the body - are able to boost the body chemistry by
As far as the cardiovascular system is concerned, according to orthomolecular nutrition, if the right building blocks (nutrients) are present in the body - in the right amounts and at the right time - the body will do the rest. In other words, if you want to lower your triglyceride levels you need to get to the root of the problem. By just pulling a dandelion out by its leaves, you are not going to get very far.
We hope you will join them. Sooner, rather than later...
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Therefore, your food choices - especially consumption of quality foods - should be given serious consideration.
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For adults, increased blood levels of Lp(a) are almost always a sign of not enough exercise.
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At Full of Health, we are sure that this simple method - so simple that, at first, its simplicity probably makes you difficult to believe - will do as much for you, as it has done for our clients and customers. If other people have benefited from Triglyceride Reduction Formula, you, or someone you know or hold dear, can benefit as well.
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We are so proud to be offering an expanded second edition of this comprehensive source of practical information, answering all questions and concerns you might have about the role triglyceride play in our health and the natural means of controlling them. Some of the important insights on lowering triglycerides you'll learn from this book include:
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So far, we have introduced Triglyceride Reduction Formula™ to our clients and customers in 42 countries: the United States (including Virgin Islands, Hawaii, and Guam), Malaysia, Australia, the United Kingdom, South Africa, Thailand, New Zealand, Germany, Belgium, Trinidad, Mexico, Italy, Spain, Pakistan, Singapore, Mauritius, Suriname (South America), France (including Martinique), Bolivia, Russia, Croatia, Poland, Portugal, Denmark, United Arab Emirates, Brunei Darussalam, Hong Kong, Macau, St. Lucia (West Indies), Norway, Saudi Arabia, Philippines, Brasil, Yemen, Kingdom of Bahrain, Turkey, China, Guatemala, Kuwait, Japan, Taiwan, and Jordan.
© 2001-2006 Reduce Triglycerides.com: Lowering High Triglycerides Naturally. Triglyceride Reduction TGs Formula™ - A Drug-Free Approach to Elevated Blood Triglycerides. All rights reserved worldwide. This document may not be copied in part or full without express written permission from the publisher. The information on reducing high triglycerides and nutrition provided herein is a general overview on this topic and may not apply to everyone, therefore, it should not be used for diagnosis or treatment of any medical condition. While reasonable effort has been made to ensure the accuracy of the information on reducing high triglycerides, Full of Health, Inc. assumes no responsibility for errors or omissions, or for damages resulting from use of the triglyceride information herein.